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Chronic Hyperinsulinaemia and Homoeopathy

Note- For well formatted article, pl. see attached file. Chronic  Hyperinsulinaemia  and Homoeopathy.pdf


Excess levels of insulin, the main energy storage hormone, circulating in the blood than expected, relative to the level of glucose is called Hyperinsulinaemia (Psora). Hyperinsulinaemia is associated with hypertension, obesity, dyslipidemia, and glucose intolerance (Psora/ Sycosis). These conditions are collectively known as ‘Metabolic syndrome’.


Chronic exposure to refined carbohydrates and simple sugars can cause elevated levels of insulin, which drives glucose levels down (Psora). This can result in hypoglycaemia (Psora). Over time, tissues may become less sensitive to insulin (Psora-Syphilis) and as a result glucose cannot enter the cells as easily. This means more glucose in the bloodstream and a greater tendency to convert it into fat instead of energy. Elevated insulin levels cause the body to have difficulty in breaking down fat too.

In type 2 diabetes, body cells become resistant to the effects of insulin (Pseudopsora). The insulin binding receptors on cells become less sensitive to insulin concentrations (Syphilis) resulting in Hyperinsulinaemia (Sycosis) and disturbances in insulin release (Psora). With a reduced response to insulin, the beta cells of the pancreas secrete increasing amounts of insulin in response to the continued high blood glucose levels resulting in Hyperinsulinaemia (Psora/ Sycosis). In insulin resistant tissues, a threshold concentration of insulin is reached causing the cells to uptake glucose and therefore decreases blood glucose levels (Psora). The high levels of insulin resulting from insulin resistance may increase insulin resistance (Syphilis).

Neonatal Hyperinsulinaemia

Hyperinsulinaemia in neonates can be due to a number of environmental and genetic factors. If the mother of the infant is a diabetic, and does not properly control her blood glucose levels, the hyperglycemic maternal blood can create a hyperglycemic environment in the foetus. To compensate for the increased blood glucose levels, foetal pancreatic beta cells can undergo hyperplasia (Sycosis). The rapid division of beta cells results in increased levels of insulin being secreted to compensate for the high blood glucose levels (Sycosis). Following birth, the hyperglycemic maternal blood is no longer accessible to the neonate resulting in a rapid drop in the newborn’s blood glucose levels (Psora). As insulin levels are still elevated this results in Hyperinsulinaemia. The Hyperinsulinaemia condition subsides after one to two days.


High levels of insulin can block stress hormones, called as catecholamines, which normally cause the release of cellular energy (Psora/ Syphilis). For normal metabolism to occur, the body needs a balanced input of insulin and catecholamines. Insulin blocks activation of the protein kinase A (PKA) enzyme (Psora/ Syphilis). After a meal, insulin levels go up (Psora), and the body stores energy primarily as triglycerides, or fat, in adipose tissue for future use. When energy is needed, catecholamine triggers activation of PKA, and energy is released by cells. But in people with Type II diabetes, the hormonal balance has been thrown off, because the body continues to produce and store more triglyceride instead of breaking down the fat as released energy (Psora/ Sycosis).

Obese people have an excess of adipose tissue which secrete various metabolites, hormones and cytokines that may play a role in causing Hyperinsulinaemia. Cytokines, especially adiponectins, secreted by adipose tissue directly affect the insulin secretion. Adiponectins are cytokines that are inversely related to percent body fat. People with low body fat have higher concentrations of adiponectins where as people with high body fat have lower concentrations of adiponectins. Hyperinsulinaemia can be due to low adiponectin concentrations in obese people.

Other causes of Hyperinsulinaemia may be Neoplasm, pancreatic cancer, PCOS and Trans Fats. (Psora/ Syphilis/ Sycosis)


There are often no noticeable symptoms of Hyperinsulinaemia except hypoglycaemia, marked by -

  • Temporary muscle weakness (Psora)
  • Brain fog (Psora)
  • Fatigue (Psora)
  • Temporary thought disorder, or inability to concentrate (Psora/ Syphilis)
  • Visual problems such as blurred vision or double vision (Psora/ Sycosis)
  • Headaches (Psora/ Syphilis/ Sycosis)
  • Shaking/Trembling (Psora)
  • Thirst (Psora/ Pseudopsora)

Other symptoms include-

Weight gain especially around the waist, producing the apple shape, not the pear shape. (Sycosis)

High systolic blood pressure (Psora/ Sycosis)

High diastolic blood pressure (Psora/ Sycosis)

High Total Cholesterol (Psora/ Sycosis)

Early male pattern baldness- Although early baldness on the top of the head may be a non-modifiable risk factor for heart disease, it may serve as a useful clinical marker to identify men at increased risk of insulin problems and cardiac risk. (Psora/ Syphilis)

Insulin resistance may play a role in the development of gout. Gout is strongly associated with the consequences of insulin resistance i.e. obesity, hypertension, hyperlipidemia and diabetes. (Psora/ Sycosis)

Hyperinsulinaemia and insulin resistance are both factors that increase the risk of developing type 2 diabetes. Hyperinsulinaemia often predates diabetes by several years. (Psora/ Syphilis)

A majority of patients with PCOS have insulin resistance and/or are obese. There is a lot of evidence that high levels of insulin contribute to increased androgen production, which worsens the symptoms of PCOS. (Psora/ Sycosis)

Risk factors

Very early puberty onset

Girls with premature puberty have been found to have elevated insulin and DHEA levels. This contributes to the weight gain usually seen in advanced stages of PCOS. (Psora/ Sycosis)

Lack of Sleep

Continued insomnia may cause body cells less sensitive to insulin which, over time, can raise the risk of obesity, high blood pressure and diabetes. Chronic sleep deprivation (under 6.5 hours per night) has the same effect on insulin resistance as aging. (Psora/ Syphilis)


Cortisol blocks the insulin receptor as its undesirable effects and contributes to insulin resistance by decreasing the rate of glucose uptake.  (Psora)

Syndrome X / Metabolic Syndrome

Syndrome X or Metabolic Syndrome is the variable combination of obesity (usually central in distribution), insulin resistance with elevated insulin levels, high blood cholesterol and hypertension. Metabolic Syndrome causes Hyperinsulinemia. (Psora/ Syphilis/ Sycosis)

Consequences of Hyperinsulinaemia

  • May lead to hypoglycemia or Diabetes mellitus type 2 (Pseudopsora)
  • Increased risk of PCOS (Psora/ Sycosis)
  • Increased synthesis of VLDL (hypertriglyceridemia) (Psora/ Sycosis)
  • Increased sodium retention by the renal tubules causing Hypertension (Psora/ Sycosis)
  • Damage to endothelial cells causing Coronary Artery Disease (Psora/ Syphilis)
  • Increased risk of cardiovascular disease (Psora/ Sycosis)
  • Weight gain and lethargy, may be due to hypothyroidism. (Psora/ Sycosis)
  • Gout / Hyperuricemia (Psora/ Sycosis)
  • Polycystic Ovary Syndrome (PCOS) (Psora/ Sycosis)


Treatment is typically achieved via diet and exercise. A low carbohydrate diet is particularly effective in reducing hyperinsulinism.

It has been shown in many studies that physical exercise improves insulin sensitivity.


Cinnamon with each meal helps keep insulin and blood sugar levels under control. The typical ½ to ¾ teaspoon dose contains a phytochemical called methyl hydroxy chalcone polymer (MHCP) which improves cellular glucose utilization and increases the sensitivity of insulin receptors in laboratory studies.  

Short Repertory of Insulin related disorders

ABDOMEN - PANCREAS; complaints of - insulin secretion decreased- cortico.

GENERALS - DIABETES MELLITUS - insulin dependent- ins. nat-p. sulph.

Toxicity - INSULIN, poisoning, ailments, from- ins. lyc. phos.

Short Repertory of Diabetes

Ankles - SWELLING, ankles - diabetes, in- arg-met.

CHEST - LUNGS; complaints of the - accompanied by – diabetes- calc-p.

CHEST - PHTHISIS pulmonalis - accompanied by – diabetes- phos.

CLINICAL - ACIDOSIS - diabetes mellitus, with- senn.

Clinical - blackness, tissues, external parts – diabetic- Ars. con. Kreos. kres. lach. Sec. solid.

Clinical - DIABETES, mellitus - acidosis, with diabetic- Ins. Nat-p.

Clinical - DIABETES, mellitus - coma, diabetic- allox. ins.

Clinical - edema, general - diabetes, mellitus, with- lac-ac.

Clinical - emaciation, general - diabetes, with- Arg-met. Ars. Ph-ac. rat. tarent. Uran-n.

Clinical - GANGRENE, general – diabetic- Ars. carb-ac. con. Kreos. kres. lach. Sec. solid.

Clinical - hyperglycemia, high blood sugar- Arg-n. Chin. Cina Ins. iod. Lyc. olnd. Phos. sacch-a. stann. verat. Zinc.

Clinical - ulcers, general – diabetic- syzyg.

Constitutions - WEAK, constitutions - diabetes, mellitus, in- arg-met. ars. carc. coca lac-ac. PH-AC. PHOS.

EXTREMITIES - GANGRENE – diabetic- ars. carb-ac. con. lach. sec. solid.

EXTREMITIES - GANGRENE - Feet – diabetic- lyc.

EXTREMITIES - PAIN - gouty - joints - diabetes, with- phase.

EXTREMITIES - PAIN - gouty - upper limbs - joints - diabetes, with- phase.

EXTREMITIES - PAIN - Lower limbs - Sciatic nerve - accompanied by - diabetes mellitus- kreos.

EXTREMITIES - PAIN - rheumatic - diabetes, in- lac-ac.

EXTREMITIES - SWELLING - Ankle - diabetes, in- arg-met.

EXTREMITIES - SWELLING - general - lower limbs - ankles - diabetes, in- arg-met.

EXTREMITY PAIN - GENERAL - rheumatic - diabetes, in- lac-ac.

EXTREMITY PAIN - JOINTS - gouty - diabetes, with- phase.

EYE - INFLAMMATION - retina – diabetic- sec.

EYES - INFLAMMATION - retina, retinitis - diabetes, in- sec.

Eyes - RETINITIS, inflammation, retina – diabetic- crot-h. phos. sec.

FEMALE - MENSES - suppressed - diabetic attack, during- uran-n.

Female - MENSES, general - ailments, menses, during - diabetes, in- uran-n.

Female - MENSES, general - suppressed - diabetic attack, during- uran-n.

FEMALE GENITALIA/SEX - MENSES - suppressed menses - diabetes; in- uran-n.

Fevers - TYPHOID, fever, salmonella – diabetes- sul-ac.

Food - APPETITE, general - ravenous, appetite, canine - diabetes, during- am-c. Coloc.

Gangrene - diabetic original- con. lach. solid.

GENERALITIES - WEAKNESS, enervation, exhaustion, prostration, infirmity - diabetes mellitus, in- Arg-met. Ars. Lac-ac.

GENERALS - BLACKNESS of external parts – diabetic- Ars. Kreos. kres. Sec.

GENERALS - DIABETES INSIPIDUS- abrom-a. acet-ac. acon. alf. all-c. am-act. ambr. apoc. arg-met. arg-mur. arg-n. ars-br. ars. Aur-m. bell. bry. cain. Cann-i. canth. caust. chinin-s. chion. chlorpr. cina Cod. conv. cortico. crat. dulc. Equis-h. eup-per. eup-pur. Ferr-m. ferr-n. gels. Glon. glyc. gnaph. gua. hell. helon. ign. indol. jab. kali-c. kali-i. kali-n. kreos. lac-ac. led. lil-t. Lith-c. lyc. mag-p. merc-c. mosch. murx. Nat-m. nicc-s. Nit-ac. nux-v. ol-an. Oxyt. ph-ac. phos. phys. pic-ac. plat-m-n. podo. puls. quas. rhus-a. samb. sang. santin. saroth. sars. sec. sel. Sin-n. squil. staph. stroph-h. Sulph. tarax. ter. thymol. thyr. uran-m. uran-n. verat-v. verb.

GENERALS - DIABETES MELLITUS - bronze diabetes- adren.

GENERALS - DIABETES MELLITUS- abrom-a. acet-ac. adren. aether alf. all-s. allox. aloe alumn. am-act. anthraco. apoc. arg-met. arg-n. arist-m. Ars-br. ars. asc-c. aspar. aur-m-n. aur. bar-m. Bor-ac. bov. brid-fr. calc-p. calc-sil. calc. canth. carb-ac. carb-v. carc. card-m. Carl. caust. cean. cephd-i. chel. chim. Chion. chlol. chlorpr. clem. coca cod. coff. coloc. con. cop. cortico. cortiso. cub. cupr-ar. cupr. cur. eup-pur. ferr-i. ferr-m. ferr-p. fl-ac. flor-p. friedr. gal-ac. galeg. glyc. Gymne. hed. helon. hydrang. hygroph-s. indgf-a. ins. Inul. iod. iris kali-act. kali-br. kali-chl. kali-i. kali-p. kiss. kreos. Lac-ac. lac-d. lach. led. lept. lith-c. lyc. lycps-v. mag-act. mag-o. mag-p. mag-s. mang-act. med. meny. merc-d. merc. moni. morind-l. morind-m. morph. mosch. mur-ac. murx. nat-ch. nat-lac. nat-m. nat-p. NAT-S. nauc-l. nep. nit-ac. nux-v. Op. orthos-s. oxyg. pancr. peps. perh. ph-ac. Phase. phlor. phos. pic-ac. pilo. plan. plb. podo. rad-br. rad-met. ran-b. rat. Rhus-a. rhus-r. rhus-t. sacch-l. sal-ac. sanic. sarcol-ac. saroth. sep. Ser-ang. sil. spong. Squil. stict. stront-c. stry-ar. sul-ac. sulfonam. sulph. syph. SYZYG. tarent. TER. Terebe. term-a. thuj. thyr. uran-m. Uran-n. Urea vanad. vichy-g. vinc-r. vince.

GENERALS - FAMILY HISTORY of - diabetes mellitus- carc. sacch. thuj.

GENERALS - INFLAMMATION - gangrenous - diabetics; in- ars. nat-pyru. sec.

GENERALS - NEUROLOGICAL complaints - accompanied by – diabetes- helon.

GENERALS - SHOCK - followed by - diabetes mellitus- op.

GENERALS - WEAKNESS - diabetes mellitus, in- acet-ac. Arg-met. Ars. carb-v. carc. con. graph. kali-c. Lac-ac. op. phos.

Glands - pancreas, general - kidneys, disease of, preceding or accompanying diabetes mellitus, or bright's disease- Phos.

Impotency - diabetes, with- coca mosch. ph-ac.

Itching - diabetes, in- mang.

Joints - ACHING, pain - diabetes, in- rat.

KIDNEYS - COMPLAINTS of kidneys - accompanied by – diabetes- saroth.

Kidneys - PAIN, kidneys - diabetes, in- ph-ac. phos.

Kidneys - SORE, pain - diabetes, in- rat.

Kidneys - WEAK, kidneys - diabetes, with- Phos.

Limbs - GANGRENE, limbs – diabetic- carb-ac. con. lach. sec. solid.

Liver - ENLARGED, liver - diabetes, in- Nat-s.

Liver - SHARP, pain - diabetes, in- sul-ac.

Liver - TENDER - diabetes, mellitus, in- kali-br.

Male - ERECTIONS, penis, troublesome - incomplete - diabetes, with- coca mosch. ph-ac.

MALE - ERECTIONS, troublesome - incomplete - diabetes, with- coca mosch. ph-ac.

MALE - ERECTIONS, troublesome - wanting, impotency - diabetes, with- HELON. mosch.

Male - IMPOTENCY, sexual - diabetes, with- coca Helon. mosch. ph-ac.

Male - SEX, male - decreased, desire - diabetes, in- coca Cupr.

MALE - SEXUAL - desire - diminished - diabetes, in- Cupr.

MALE GENITALIA/SEX - ERECTIONS - wanting - diabetes, with- acon. cann-s. coca con. cupr. eup-pur. Helon. kali-c. mosch. ph-ac. sulph.

MALE GENITALIA/SEX - SEXUAL DESIRE - diminished - diabetes; in- Cupr.

Menses - absent, suppressed, amenorrhoea - diabetes, in- uran-n.

MIND - ALCOHOLISM - diabetes; with- med. nux-v.

MIND - ALCOHOLISM, dipsomania - diabetes, with- med.

MIND - ANXIETY - diabetes; in- cod. Nat-s.

Mind - ANXIETY, general - diabetes, in- arg-n. cod. Nat-s. Phos.

MIND - COMA - diabetes; in- alum. ars. carb-v. carbn-o. cur. op.

Mind - DEPRESSION, sadness - diabetes, with- helon. lyc. nat-s. op.

MIND - DULLNESS - diabetes, in- Helon. Op. ph-ac. sul-ac.

Mind - DULLNESS, mental - diabetes, in- acet-ac. Helon. Op. ph-ac. phos. sul-ac.

MIND - DULLNESS, sluggishness, difficulty of thinking and comprehending - diabetes, in- acet-ac. HELON. NAT-S. OP. sul-ac.

MIND - FEAR - diabetes, in- cod. NAT-S.

MIND - FEAR - sudden - followed by - diabetes mellitus- op.

Mind - FEARS, phobias,general - diabetes, in- cod. Nat-s. Phos.

MIND - GRIEF - diabetes; with- aur-m-n. aur. ign. mag-m. nat-s. ph-ac. tarent.

MIND - IRRITABILITY - diabetes, in- Helon. Lycps-v. Nux-v.

Mind - IRRITABILITY, general - diabetes, in- Helon. Lycps-v. Nux-v.

MIND - MEMORY - weakness of memory - diabetes; in- kali-br. lyc. nux-m. nux-v. ph-ac.

MIND - MEMORY - weakness, loss of - diabetes, in- OP.

Mind - MEMORY, weakness, of - diabetes, in- lyc. Op. phos.

MIND - PROSTRATION of mind, mental exhaustion, brain fag - diabetes, in- NAT-S.

MIND - RESTLESSNESS – diabetic- helon.

MIND - SADNESS - diabetes; during- Helon. Nat-s. Op.

MIND - SADNESS, despondency, depression, melancholy - diabetes, in- Helon. Nat-s. Op.

Mouth - CLAMMY, mouth - diabetes, in- uran-n.

Pulse - FAST, pulse, elevated, exalted - diabetes, in, 90, relieved- uran-n.

Pulse - SLOW, pulse - diabetes, in- Op.

Pulse - SMALL, pulse - diabetes, in- uran-n.

Pulse - WEAK, pulse - diabetes mellitus, in- kali-br.

RECTUM - CONSTIPATION - diabetes mellitus, with- symph.

Retina - inflammation – diabetic- sec.

SKIN - GANGRENE, from burns or gangrenous sores – diabetic- ARS. carb-ac. con. echi. KREOS. kres. lach. SEC. solid.

SKIN - ITCHING - diabetes, in- agar. calad. mang.

SKIN - ITCHING - diabetes, in- mang.

SKIN - ITCHING - diabetics; in- Cephd-i.

Skin - ITCHING, skin - diabetes, in- mang.

SKIN - ULCERS - diabetes, in- syzyg.

SKIN - ULCERS – diabetic- syzyg.

Sleep - INSOMNIA, sleeplessness - diabetics, in- carc. coca Uran-n.

SLEEP - SLEEPLESSNESS - diabetics, in- Uran-n.

SLEEP - SLEEPLESSNESS - general - diabetics, in- uran-n.

STOMACH - APPETITE - ravenous, canine, excessive - emaciation, with - diabetes, during- am-c. Coloc.

STOMACH - THIRST - extreme - diabetes mellitus, with- sat-h.

TEETH - Aggravation - cough - diabetes in- sec.

TEETH - CARIES, decayed, hollow - diabetes mellitus- sul-ac.

TEETH - CARIES, decayed, hollow - general - diabetes, in- sul-ac.

TEETH - COUGH agg. - diabetes; in- sec.

Teeth - decay, caries; hollow - diabetes, in- sul-ac.

Urine - PROFUSE, increased, urine - diabetes, with- acet-ac. Phos.

Vision - DIM, vision - diabetes, in- phos. tab. tarent.

Vision - DIM, vision - dull - diabetes, in- sul-ac.

Weakness - DIABETES, mellitus, weakness, in- alf. Arg-met. Ars. carc. coca Lac-ac. PH-AC. PHOS.




 Encyclopedia Homoeopathica

Endocrine System Chapter 103. Normal Pregnancy > Endocrine System Tintinalli's Emergency Medicine

Relation to Insulin Resistance" border="0" height="44" width="34"> Chapter 11. Cardiovascular Disorders: Vascular Disease > Relation to Insulin Resistance Pathophysiology of Disease, 6e

Etiology and Pathogenesis" border="0" height="43" width="32"> Chapter 151. Diabetes Mellitus and Other Endocrine Diseases > Etiology and Pathogenesis Fitzpatrick's Dermatology in General Medicine, 8e

Pathogenesis" border="0" height="45" width="35"> Chapter 16. Disproportionate Fetal Growth > Pathogenesis CURRENT Diagnosis & Treatment: Obstetrics & Gynecology, 11e

Acanthosis Nigricans" border="0" height="45" width="34"> Chapter 17. Polycystic Ovarian Syndrome and Hyperandrogenism > Acanthosis Nigricans Williams Gynecology, 2e

Clinical Findings" border="0" height="48" width="34"> Chapter 18. Gynecologic Disorders > Clinical Findings CURRENT Medical Diagnosis & Treatment 2014

Clinical Presentation" border="0" height="42" width="32"> Chapter 18. Hypoglycemic Disorders > Clinical Presentation Greenspan’s Basic & Clinical Endocrinology, 9e

Congenital Hyperinsulinism" border="0" height="43" width="33"> Chapter 18. Hypoglycemic Disorders > Congenital Hyperinsulinism Greenspan’s Basic & Clinical Endocrinology, 9e

Diagnosis" border="0" height="46" width="36"> Chapter 18. Hypoglycemic Disorders > Diagnosis Greenspan’s Basic & Clinical Endocrinology, 9e

Outcome" border="0" height="47" width="36"> Chapter 18. Hypoglycemic Disorders > Outcome Greenspan’s Basic & Clinical Endocrinology, 9e

Persistent Hyperinsulinism" border="0" height="43" width="33"> Chapter 18. Hypoglycemic Disorders > Persistent Hyperinsulinism Greenspan’s Basic & Clinical Endocrinology, 9e

Transient Hyperinsulinism" border="0" height="43" width="33"> Chapter 18. Hypoglycemic Disorders > Transient Hyperinsulinism Greenspan’s Basic & Clinical Endocrinology, 9e

Treatment" border="0" height="48" width="37"> Chapter 18. Hypoglycemic Disorders > Treatment Greenspan’s Basic & Clinical Endocrinology, 9e

Body Mass Index" border="0" height="48" width="36"> Chapter 18. Pancreatic Cancer > Body Mass Index The MD Anderson Manual of Medical Oncology, 2e

Hyperinsulinemia-Euglycemia Therapy" border="0" height="43" width="... Chapter 188. ß-Blockers > Hyperinsulinemia-Euglycemia Therapy Tintinalli's Emergency Medicine

Hyperinsulin/Euglycemia Therapy" border="0" height="47" width="35"> Chapter 189. Calcium Channel Blockers > Hyperinsulin/Euglycemia Therapy Tintinalli's Emergency Medicine

Intrauterine Growth-Restricted Infants" border="0" height="44" widt... Chapter 2. The Newborn Infant > Intrauterine Growth-Restricted Infants CURRENT Diagnosis & Treatment: Pediatrics, 21e

Insulin-Sensitizing Agents" border="0" height="45" width="34"> Chapter 20. Treatment of the Infertile Couple > Insulin-Sensitizing Agents Williams Gynecology, 2e

Nonpharmacologic" border="0" height="51" width="39"> Chapter 220. Acanthosis Nigricans > Nonpharmacologic The Color Atlas of Family Medicine, 2e

Effect of Preexisting Type I and Type II Diabetes on Pregnancy" bor... Chapter 223. Common Medical Problems in Pregnancy > Effect of Preexisting Type I and Type II Diabetes on Pregnancy Principles and Practice of Hospital Medicine

Hyperuricemia and Metabolic Syndrome" border="0" height="46" width=... Chapter 359. Disorders of Purine and Pyrimidine Metabolism > Hyperuricemia and Metabolic Syndrome Harrison's Online

Metabolic Effects" border="0" height="46" width="35"> Chapter 39. Thyroid and Anti-Thyroid Drugs > Metabolic Effects Goodman & Gilman's The Pharmacological Basis of Therapeutics, 12e

Glucose, Insulin, and Fetal Macrosomia" border="0" height="47" widt... Chapter 4. Fetal Growth and Development > Glucose, Insulin, and Fetal Macrosomia Williams Obstetrics, 23e

Response to Treatment" border="0" height="47" width="36"> Chapter 4. Hypothalamus and Pituitary Gland > Response to Treatment Greenspan’s Basic & Clinical Endocrinology, 9e

Increased Cancer Risk" border="0" height="45" width="35"> Chapter 41. Pancreatic Hormones & Antidiabetic Drugs > Increased Cancer Risk Basic & Clinical Pharmacology, 12e

Rationale" border="0" height="47" width="35"> Chapter 51. Preventive Strategies for Coronary Heart Disease > Rationale Hurst's The Heart, 13e

Diet" border="0" height="50" width="35"> Chapter 6. Dermatologic Disorders > Diet CURRENT Medical Diagnosis & Treatment 2014

Does Insulin Resistance Cause Hypertension Independent of Hyperinsu... Chapter 69. Pathophysiology of Hypertension > Does Insulin Resistance Cause Hypertension Independent of Hyperinsulinemia? Hurst's The Heart, 13e

What Is the Role of Metabolic Syndrome or Insulin Resistance in Pri... Chapter 69. Pathophysiology of Hypertension > What Is the Role of Metabolic Syndrome or Insulin Resistance in Primary Hypertension? Hurst's The Heart, 13e

Long-Term Effects" border="0" height="49" width="33"> Chapter 7. Endocrine Pancreas > Long-Term Effects Endocrine Physiology, 4e

Insulin resistance and type 2 diabetes mellitus" border="0" height=... Chapter 77. Biology of Obesity > Insulin resistance and type 2 diabetes mellitus Harrison's Online

Polycystic Ovarian Syndrome" border="0" height="50" width="39"> Chapter 9. Abortion > Polycystic Ovarian Syndrome Williams Obstetrics, 23e

Effects on Adipose Tissue" border="0" height="46" width="35"> Chapter 9. Glucocorticoids and Adrenal Androgens > Effects on Adipose Tissue Greenspan’s Basic & Clinical Endocrinology, 9e

Laboratory Findings" border="0" height="44" width="34"> Chapter 9. Glucocorticoids and Adrenal Androgens > Laboratory Findings Greenspan’s Basic & Clinical Endocrinology, 9e

Summary" border="0" height="45" width="35"> Chapter 9. Glucocorticoids and Adrenal Androgens > Summary Greenspan’s Basic & Clinical Endocrinology, 9e

Risk Factors for Atherosclerosis" border="0" height="52" width="35"... Section 17. Skin Signs of Vascular Insufficiency > Risk Factors for Atherosclerosis Fitzpatrick's Color Atlas and Synopsis of Clinical Dermatology, 7e

Etiology and Pathogenesis" border="0" height="51" width="34"> Section 5. Miscellaneous Epidermal Disorders > Etiology and Pathogenesis Fitzpatrick's Color Atlas and Synopsis of Clinical Dermatology, 7e

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Comment by Dr Rajneesh Kumar Sharma MD(Hom) on September 7, 2014 at 2:17pm

That's is absolutely correct Dr. Rafeeque.

I do also face such problems.


Comment by Dr Muhammed Rafeeque on September 7, 2014 at 10:34am

The difficulty in managing such cases is, the moment they get normal report, they stop the consultation. And finally coming with some complications later. Even though they do not come to us regularly, they are entitled as our patient among the family circle, and we will be blamed for the complications. So, I always take precautions in such cases - "If you want to come, come regularly, or else do not come"

Comment by Dr Rajneesh Kumar Sharma MD(Hom) on August 21, 2014 at 2:57am

Thanks a lot of sir. Regards

Comment by Kuldip Singh on August 21, 2014 at 2:48am

Homeopaths generally find it not easy to resolve cases of diabetes. It all depends on close repertorisation of an individual case of diabetes. In this context Dr Rajnish Kumar Sharma has provided very relevant and useful reportorial rubrics. He certainly deserves all appreciation for compiling this interesting blog.

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