Creating Waves of Awareness
A very affectionate child, 4.5 years of age, having symptoms of Duchenne Muscular Dystrophy came to me on 16 Feb 2010, with tremendous weakness in muscles of upper and lower limbs with hypertrophy.
Due to weakness in the limbs, this child wanted to be carried after walking for a little distance, and felt very sensitive to pain. He was not able to climb up and downstairs on his own, otherwise he requires support.
His CPK enzyme levels were 33616 IU/L on 16/09/09. (Normal levels are 25-192 IU/L).
He was on Prednisolone 40mg/day on a tapering dose for a 6 months period down to 10mg/day without any improvement in the weakness. In fact, his muscle weakness had increased. He has flat feet.
His nature was very affectionate; restless; shy; likes to play in company; does not like to go to school; when questioned, he becomes dull, unable to answer either due to timidity or difficulty on thinking; getting irritable on very small matters; fears dark; with active memory; found to be disobedient, biting his nails and putting the fingers in mouth, continuously moving here and there in the consulting room.
His mother told me that he was a homesick, moody child but desires to travel a lot. He eats pencil a lot. He sleeps on abdomen. He still wets his bed at night.
Neonatal Jaundice at birth
H/O Partially developed Chickenpox 2 years back
Diabetes: PGF & MGF
Hypertension: PGF & MGF
Myocardial Infarction: PGF
Desires: Indigestible things++
Duchenne muscular dystrophy
• Duchenne muscular dystrophy (DMD) is a recessive X-linked form of muscular dystrophy, which results in muscle degeneration, difficulty in walking & breathing, and death.
• The incidence is around 1 in 3,600 boys.
• Females and males are affected, though females are rarely affected and are more often carriers.
• The disorder is caused by a mutation in the dystrophin gene, located in humans on the X chromosome (Xp21).
• The dystrophin gene codes for the protein dystrophin, an important structural component within muscle tissue.
• Dystrophin provides structural stability to the dystroglycan complex (DGC), located on the cell membrane.
• Symptoms usually appear in male children before age 5 and may be visible in early infancy. Progressive proximal muscle weakness of the legs and pelvis associated with a loss of muscle mass is observed first. Eventually this weakness spreads to the arms, neck, and other areas.
• Early signs may include pseudohypertrophy (enlargement of calf and deltoid muscles), low endurance, and difficulties in standing unaided or inability to ascend staircases.
• As the condition progresses, muscle tissue experiences wasting and is eventually replaced by fat and fibrotic tissue (fibrosis).
• By age 10, braces may be required to aid in walking but most patients are wheelchair dependent by age 12.
• Later symptoms may include abnormal bone development that lead to skeletal deformities, including curvature of the spine. Due to progressive deterioration of muscle, loss of movement occurs, eventually leading to paralysis.
• The average life expectancy for patients afflicted with DMD is around 25, but this varies from individual to individual.
Pathogenesis of DMD
• Dystrophin is responsible for connecting the cytoskeleton of each muscle fiber to the underlying basal lamina (extracellular matrix) through a protein complex containing many subunits.
• The absence of dystrophin permits excess calcium to penetrate the sarcolemma (cell membrane). Alterations in these signaling pathways cause water to enter into the mitochondria which then burst.
• Muscle fibers undergo necrosis and are ultimately replaced with adipose and connective tissue.
2. Sensitive , oversensitive, Pain to
3. Extremities, Weakness, Upper limb
4. Extremities, Weakness, Lower limb
5. Restlessness, Children, in
7. Company, desire for
8. Aversion, School, to
9. Dullness, Children, in
10. Irritability, Children, in
11. Fear, Dark
12. Memory, Active
14. Biting, Nails
15. Mouth, Fingers in mouth, Children put
17. Mood, Changeable, variable
18. Travel, Desire to
19. 19. Generalities, Food, Indigestible things, Desire
20. Sleep, Position, Abdomen, on
21. Bladder, Urination, Involuntary, Night, incontinence in bed
Result of Repertorisation:
Calc phos: 32/21
Calc carb: 31/18
Calc Phos 6x tds for 10 days
Prednisolone stopped by the patient’s parents a week back as there was no relief.
Follow Up (26/02/10)
He suffered from mumps on right side on the 20th Feb and recovered on his own within 4 days without any medicine.
Playing with friends, weakness decreased a lot after the mumps.
Remedy continued in bd doses for 15 days.
I already advised them that he might suffer from any type infectious disease after starting the treatment based on my understanding from "Predictive Homoeopathy." So, if anything happens, please, don’t give any medicine. (Ref: Verbatim by Dr Praful Vijaykar, Chapter 14)
• Why the Mumps did came up? Is it a good or bad sign?
• Mumps is a tumor virus like HSV, EBV & CMV.
• Why are they known as a tumor virus in the allopathic world? Because when they infect and go to the cell, they cause a change in mitotic cycle in such a manner that extra growth occurs in a cell. The cell starts growing excessively giving rise to tumors. For us it is a Sycotic virus.
• Therefore, sycotic virus being thrown out of the body is a good sign in this case. The tumor viruses when thrown out to clear the internal malady or dysfunction are exteriorization....
• So, just rejoice and lay your hands off if a tumor virus appears. The virus is in the neuro-ectoderm and is thrown off. This is a excellent sign. These viruses, especially tumor viruses harboring in the nerves or muscles are thrown off through the prochordal plate. So, the patient will first get some throat problem and then symptoms on the body.
• Tumor viruses being exteriorized is one of the best result for the Homoeopath.
• (Ref: Verbatim by Dr Praful Vijaykar, Chapter 14)
Follow Up (12/03/10)
Weakness in limbs much decreased.
He started running as well as climbing staircase without support.
He was getting thick nasal discharge since week with occasional dry cough.
His confidence level increased.
Remedy continued in od dose for 1 month.
Follow Up (12/04/10)
He is walking without asking for carrying. He is playing cricket, running here & there, climbing up & down stairs without any support. Now he is found as very happy looking child.
Remedy continued in od dose till 19/01/11 when his CPK enzyme levels were measured and found to be 1075 IU/L.
Follow Up (19/01/11)
His weakness in the limbs was absent.
His bed wetting also stopped.
He complained of Pain in his knees while squatting.
Otherwise, he is playing, running, climbing up, and downstairs without any support.
Remedy Prescribed: Calc Phos 30 3 doses followed by Placebo daily.
Recent Follow Up (20/10/11)
Weakness in the limbs absent.
No bed wetting.
Pain in his knees while squatting was absent.
He is playing, running, climbing up & downstairs without any support enjoying his life.
He is on Placebo.
Dr Prasad Rasal HMD (London), MD (Hom)
Sai Swami Computerized Homoeopathic Clinic, Hospital, Research Center and Maternity Home
Bravo! Have you had more cases of muscular dystrophy? This look into the development of a young child's recovery from a typically costly and devastating disease to growth and affecting an entire family came to abrupt halt due to well chosen homeopathic remedy selection.
Thank you Debby for a very prompt reply.
I have few more cases which I'll share as time permits.
Nice Case! Congratulations!!!
Verbatim is a good book to learn and apply Predictive Homoeopathy's teaching.
Also interesting that you chose Tissue Remedy...instead of 30C or 200C. What was your thought process for this?
Dear Sir, Thanks a lot for your valuable post on Musculer Dystrophy. I'm sure this is going to help & inspire me and many others for treating this type of patients.
Sir, miraculous cure and unbelievable knowledge... Regards...
Thank you Dr Rajneesh
Keep it up...
EXCELLANT CASE OF MUSCULAR DYSTROPHY.ITS an eye opener how one single remedy cured such a serious case and saved a child. WELL DONE DR. PRASAD RASAL.
thanks for good sharing.
Thank You Dr Bajwaji...
Dear Dr Rasal,
Wonderful brilliant patient case. Unbelievable what can be achieved when matching Rx with the case at hand. Pure 'medicine qua medicine'.
Thanks again, Herman Jeggels
Good one sir.. perfect approach! Thank you for sharing
I have a case of DMD, boy aged 5yrs, with Gower's positive. Before the treatment, CPK was 32,020 u/l and CKMB 190.
Right calf muscle was affected. Child also had history of recurrent URTI and pin worm infestations. After case analysis remedy came up was Ambra grisea. (I considered Bashful, Artistic aptitude, Hyperactive, Destructive, Hot + Thirstless) Few months after the treatment CPK levels were 6154 and CKMB 269.
But muscle weakness is still progressing, now left calf is also affected. Stumbling and falling has increased, unable to go to his school. But his generals are good, he is free from all other complaints, even his destructive behavior has came down.
Sir how should be the prognosis of cure in DMD?